Monday, December 24, 2012

SEAL Team 4 commanding officer dies in Afghanistan

By Courtney Kube, NBC News

Updated at 4 p.m. ET: The commanding officer of SEAL Team 4 died in Afghanistan Saturday, U.S. military officials announced Sunday.?

Military officials tell NBC News they are looking into the death of Cmdr. Job W. Price as a possible suicide, but that his death remains under investigation.

Price, 42, of Pottstown, Pa., was in charge of coordinating all Team 4 missions.

Price?was in Afghanistan supporting stability operations in Uruzgan Province. He was assigned to an East Coast-based Naval Special Warfare unit based in Virginia Beach, Va.


According to his LinkedIn profile, Price had worked for the Department of Defense since 1989. He attended Pottstown Senior High School.?

SEAL Team 4 is among eight SEAL team deployments. SEAL Team 6 is best known among them for finding and then killing Osama bin Laden, who orchestrated the 9/11 attacks on America.??

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Source: http://usnews.nbcnews.com/_news/2012/12/23/16109338-seal-team-4-commanding-officer-dies-in-afghanistan?lite

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News digest ? cancer risk, prostate genes, melanoma, immune ...

UK newspapers

It?s our final news digest of 2012

Thankfully (at least as we write this) the world hasn?t ended, so we?re able to welcome you to our final news digest of 2012.

  • We start with our big story this week: despite improved survival, boys born in 2027 will have a 50 per cent chance of developing cancer at some point in their lives ? up from 44 per cent among men today. This was covered widely in the press and on TV (here?s the BBC?s take), while we discussed the underlying reasons in this blog post. We released the stats to highlight our new TV advert, which you can watch below:
  • Here?s a fantastic example of research taking things forward. Researchers at The Institute of Cancer Research discovered rare genetic flaws in the immune system ? rather than the cancer itself ? that are involved in breast and ovarian cancer. Here?s our coverage of this completely new concept.
  • Researchers at our Cambridge Research Institute made an intriguing step forward in understanding advanced prostate cancer. Using tissue from patients, rather than lab-grown prostate cancer ?cell lines?, they found a completely new gene network driving the disease. Here?s our press release, and you can read about the study in this in-depth blog post (which also features a nifty little video).
  • Drug resistance is a big problem in cancer treatment, particularly the new-generation of ?targeted? melanoma therapies. But as we reported on Thursday, researchers in Manchester think they?ve found how resistance to experimental drugs called MEK inhibitors can develop ? and this is before the drugs have even been through clinical trials.
  • Speaking of targeted therapies, the Daily Mail had this excellent, balanced feature about a woman with lung cancer who?s?benefiting?from a new drug called crizotinib. In case you missed it, here?s our comment piece on the promise and challenges of this new era of cancer treatment. ?
  • There?s been a lot in the news about radiotherapy and its side effects recently.?We?ve?been campaigning for better access to newer, better-targeted forms of this important cancer treatment. So we were delighted to hear this week that the Government has boosted its new ?Radiotherapy Innovation Fund?, from ?15m to ?23m. This is a fantastic early Christmas present for everyone who helped us with our Voice for Radiotherapy campaign.
  • The latest NHS Bowel Cancer Audit was published on Monday. The good news: things are continuing to improve. The bad news: far too many people are being diagnosed as an emergency case. Here?s our news story.
  • Researchers at our Beatson Institute in Glasgow discovered that cancer cells that contain a faulty protein called p53 have difficulty growing without an essential amino acid called serine (here?s our press release). P53 ? discovered by our scientists in the 1970s ? is one of the most important proteins in cancer, but exploiting it has so far eluded researchers. We hope this new discovery will begin to change that.
  • GPs have a skin cancer ?blackspot? and are failing to spot patients with melanoma, according a story in the Daily Telegraph. We felt that ?blind spot? may have made a better headline, although we agree that earlier diagnosis can make a huge difference in this disease.
  • A tricky one this ? a new study of healthcare data found that people whose parents get cancer at an old age are more likely than previously suspected to get cancer themselves. Should we be worried? No. The size of the effect is small and the study didn?t take a variety of factors into account, as we point out in our comment at the end of this Telegraph article.
  • Early lab research is showing promise in mice, according to the BBC, who report that a new form of ?Trojan Horse? immunotherapy can ?completely eliminate the disease? in laboratory animals.
  • The EU is planning tougher warnings on cigarette packs according to the BBC, while the Guardian highlights the proposals to ban menthol cigarettes and other flavoured varieties. This is good to hear, although for us the priority is to put tobacco products in plan, standardised packaging. If you haven?t already done so, sign our petition.
  • Macmillan?s high profile story on Thursday warned of ?ageism? in the NHS, and that older cancer patients may be missing out on treatments that could help them. Here?s the BBC?s coverage.
  • We spotted this US perspective on so-called ?Big Data? in healthcare, by the CEO of a healthcare data company whose father has prostate cancer.
  • The Independent had this excellent interview with Professor Sir Paul Nurse, our Nobel Prize-winning former CEO who?s heading up the new Crick Institute in London.
  • Could a joint supplement called?glucosamine help people live longer? The evidence from this study, reported in the Telegraph, suggests it could, but we?re sceptical ? the study is far from conclusive

And finally?

  • It?s a headline-writer?s dream, but something of a nightmare for balanced cancer reporting. US researchers discovered that applying physical pressure to breast cancer cells grown in the lab could encourage them to ?revert? and become less cancer-like. And so, with tedious and ? in many cases slightly chauvinistic ? predictability, began a deluge of stories reporting that ?squeezing breasts could prevent cancer?. No it can?t, and as office party season winds to a close, we?d like to issue a semi-serious warning. Ladies, if your drunken colleague offers to help alter your cancer risk, point him to this, more balanced take, and then politely move away.

So, that?s it for 2012 ? a fantastic year for us here at CRUK and for progress against cancer. In case you missed it, here?s our interactive review of the year?s progress. Thanks to all of you for your support and comments ? see you in 2013.

Henry and Olly

This entry was posted in Cancer in the news, Cancer Research UK-funded research, News digest by Henry Scowcroft. Bookmark the permalink.

Source: http://scienceblog.cancerresearchuk.org/2012/12/22/news-digest-cancer-risk-prostate-genes-melanoma-immune-system-faults-and-more/

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Sunday, December 23, 2012

Amazon Deal of the Day - Free One-Day Shipping! (Little green footballs)

Share With Friends: Share on FacebookTweet ThisPost to Google-BuzzSend on GmailPost to Linked-InSubscribe to This Feed | Rss To Twitter | Politics - Top Stories Stories, RSS and RSS Feed via Feedzilla.

Source: http://news.feedzilla.com/en_us/stories/politics/top-stories/272589844?client_source=feed&format=rss

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Family Loses Home Right Before the Holidays | 9 On Your Side

Midway Park, N.C. ? A family loses their home a few days before Christmas.

Just before 1 a.m. Saturday morning the Piney Green Volunteer Fire Department responded to a mobile home fire at1138 Rocky Run Rd in Midway Park, N.C.

Chief Roy Hall says when they arrived on scene the right side of the house was already gone and the home wasn?t able to be saved. The fire department says the home was a total loss.

The family was able to escape the fire without any injuries. No word yet on what caused on the fire.

Hall says the Red Cross did assist with finding the family a place to stay.

Source: http://www2.wnct.com/news/2012/dec/22/family-loses-home-right-holidays-ar-2880656/

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candida carnation: Biology Syllabus ? So What Does The Subject ...

If you?re encountered with biology syllabus assessment in that case he/she really needs to fully grasp the exact objectives to make sure that he/she are able to comprehend the objective. There are 3 well-known objectives concerning learning the field of biology which generally are to have practical knowledge with understanding, ability to actually deal with important information and resolve issues in conjunction with learning experimental skillsets as well as inspections.

The actual Biology Syllabus entails of scientific phenomena specifics hence these particular 3 important objectives are crucial for the main assessment procedure. A few other reasons for assessment are to be capable of making predictions, address issues, manipulate data and locate ideal sources to get knowledge acquisition.

19 December 2012 | Reference and Education

Source: http://www.apodimosstudents.com/reference-and-education/biology-syllabus-so-what-does-the-subject-focused-on/

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Source: http://otvtekod.posterous.com/biology-syllabus-so-what-does-the-subject-foc

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Source: http://candida-carnation.blogspot.com/2012/12/biology-syllabus-so-what-does-subject.html

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Saturday, December 22, 2012

Boehner says Congress, Obama must keep seeking "fiscal cliff" deal

WASHINGTON (Reuters) - House Speaker John Boehner said on Friday congressional leaders and President Barack Obama must try to move on from Republicans' failed tax plan and work together to avert the looming U.S. "fiscal cliff."

Boehner, the top Republican in Congress, was unrepentant at a Capitol Hill news conference the day after the stunning failure of his "Plan B" option, a development that raised doubts about his own leadership and left negotiations in disarray.

The failure cast fresh uncertainty over talks to avoid roughly $600 billion in across-the-board tax hikes and automatic government spending cuts due to begin in January that could push the U.S. economy back into recession in 2013. This combination of tax hikes and spending cuts is known as the "fiscal cliff."

It was "not the outcome I wanted," Boehner said after he was unable to muster enough Republican support in the House for a plan that would have limited income-tax increases to only the wealthiest sliver of the population - those earning $1 million and more.

Boehner, Obama's chief negotiating partner in talks seeking a bipartisan "fiscal cliff" solution, said he was not concerned that Thursday's withdrawn vote threatened his position as House speaker. He did not outline a clear path forward on negotiations.

The speaker failed to control unruly conservative fellow House Republicans who adamantly oppose raising taxes on anyone. They delivered a stinging rebuke to Boehner by refusing to back his "Plan B" approach.

Boehner said a divided Washington must come together to revamp the massive U.S. tax code in a way that helps spur economic growth. "How we get there, God only knows," he said.

Stocks dropped sharply at the open of trading, one day after Boehner's bill was put on the shelf.

Investors have been increasingly optimistic that a deal would be worked out before the end of the year, but this has changed that equation. Major indexes lost more than 1 percent, though stocks are still higher for the week, suggesting investors still hold out hope that an agreement will be brokered in Washington.

'SEE YOU LATER'

Boehner had hoped to use the Plan B option to pressure Obama in "fiscal cliff" talks. But late on Thursday he abruptly pulled the legislation. House members, heading to their home states for the holidays, were instructed to be available on 48 hours notice if necessary.

"They went from Plan B to plan see-you-later," Obama adviser David Axelrod said on MSNBC on Friday morning.

Obama said he still plans to work with Congress and was hopeful for a bipartisan solution, his press secretary said in a statement late on Thursday. The House is controlled by Republicans and the Senate by Obama's fellow Democrats.

The crumbling of Boehner's plan highlights his struggle to lead some House Republicans who flatly reject any deal that would increase taxes on anyone.

Republican Representative Tim Huelskamp criticized Boehner's handling of the negotiations, saying the speaker had "caved" to Obama opening the door to tax hikes. Huelskamp, a dissident first-term congressman, said he was not willing to compromise on taxes even if they are coupled with cuts to government spending sought by conservatives.

Conservatives "are so frustrated that the leader in the House right now, the speaker, has been talking about tax increases. That's all he's been talking about," Huelskamp said on MSNBC on Friday morning.

"There's been very little outreach by this leadership team to conservatives. ... Do not ask for tax increases. We're not going to give them," Huelskamp said, added: "We can still get this done."

Obama and his fellow Democrats in Congress are insisting that the wealthiest Americans pay more in taxes in order to help reduce high federal budget deficits and avoid deep spending cuts.

SENATE LEGISLATION

Democrats are now stepping up efforts to gather some Republican votes for a Democratic bill passed by the Senate months ago that would extend the expiring tax cuts to all but the wealthiest Americans.

"What we'll have to do is figure out where that line is that gives us those 218 votes" needed to garner a majority of the House behind legislation, Republican Representative Michael Burgess said on CNBC on Friday.

Senate Republican Leader Mitch McConnell, who has experience helping to forge deals when House Republicans are in disarray, is likely to play a larger role now in attempting to rescue the situation along with other Senate Republicans, who have been more receptive to compromising.

Wall Street and numerous chief executives have expressed frustration with Washington and the wrangling over a deal that has persisted for weeks with little progress.

Aetna Inc Chief Executive Officer Mark Bertolini said he feels negotiations seem like they are falling apart, calling the back-and-forth among politicians "pitiful and embarrassing" in the Wall Street Journal on Friday.

"The worst-case scenario is increasingly becoming a probable scenario," BlackRock Inc Chief Executive Officer Laurence Fink told the newspaper.

Sean West, a Eurasia Group analyst, told Reuters: "Boehner didn't have a ton of good moves, and has even fewer now." He said he thought Boehner was "either headed back to the White House for a big deal or he's accepting whatever bipartisan fallback is crafted by Senate leaders. Hard to see him coming back with another partisan gambit."

(Additional reporting by Richard Cowan and Susan Heavey; Editing by Fred Barbash and Will Dunham)

Source: http://news.yahoo.com/republicans-push-own-fiscal-cliff-plan-talks-frozen-005408461--business.html

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Transplanted neural stem cells treat ALS in mouse model

Thursday, December 20, 2012

Amyotrophic lateral sclerosis (ALS), also known as Lou Gehrig's disease, is untreatable and fatal. Nerve cells in the spinal cord die, eventually taking away a person's ability to move or even breathe. A consortium of ALS researchers at multiple institutions, including Sanford-Burnham Medical Research Institute, Brigham and Women's Hospital, and the University of Massachusetts Medical School, tested transplanted neural stem cells as a treatment for the disease. In 11 independent studies, they found that transplanting neural stem cells into the spinal cord of a mouse model of ALS slows disease onset and progression. This treatment also improves host motor function and significantly prolongs survival. The transplanted neural stem cells did not benefit ALS mice by replacing deteriorating nerve cells. Instead, neural stem cells help by producing factors that preserve the health and function of the host's remaining nerve cells. They also reduce inflammation and suppress the number of disease-causing cells in the host's spinal cord. These findings, published December 19 in Science Translational Medicine, demonstrate the potential neural stem cells hold for treating ALS and other nervous system disorders.

"While not a cure for human ALS, we believe that the careful transplantation of neural stem cells, particularly into areas that can best sustain life?respiratory control centers, for example?may be ready for clinical trials," Evan Y. Snyder, M.D., Ph.D., director of Sanford-Burnham's Stem Cell and Regenerative Biology Program and senior author of the study.

Neural stem cells

In this study, researchers at multiple institutions conducted 11 independent studies to test neural stem cell transplantation in a well-established mouse model of ALS. They all found that this cell therapy reduced the symptoms and course of the ALS-like disease. They observed improved motor performance and respiratory function in treated mice. Neural stem cell transplant also slowed the disease's progression. What's more, 25 percent of the treated ALS mice in this study survived for one year or more?roughly three to four times longer than untreated mice.

Neural stem cells are the precursors of all brain cells. They can self-renew, making more neural stem cells, and differentiate, becoming nerve cells or other brain cells. These cells can also rescue malfunctioning nerve cells and help preserve and regenerate host brain tissue. But they've never before been studied extensively in a good model of adult ALS.

How neural stem cells benefit ALS mice

Transplanted neural stem cells helped the ALS mice, but not for the obvious reason?not because they became nerve cells, replacing those missing in the ALS spinal cord. The biggest impact actually came from a series of other beneficial neural stem cell activities. It turns out neural stem cells produce protective molecules. They also trigger host cells to produce their own protective molecules. In turn, these factors help spare host nerve cells from further destruction.

Then a number of other positive events take place in treated mice. The transplanted normal neural stem cells change the fate of the host's own diseased neural stem cells?for the better. This change decreases the number of toxin-producing, disease-promoting cells in the host's spinal cord. Transplanted neural stem cells also reduce inflammation.

"We discovered that cell replacement plays a surprisingly small role in these impressive clinical benefits. Rather, the stem cells change the host environment for the better and protect the endangered nerve cells," said Snyder. "This realization is important because most diseases are now being recognized as multifaceted in their cause and their symptoms?they don't involve just one cell type or one malfunctioning process. We are coming to recognize that the multifaceted actions of the stem cell may address a number of these disease processes."

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Sanford-Burnham Medical Research Institute: http://www.burnham-inst.org

Thanks to Sanford-Burnham Medical Research Institute for this article.

This press release was posted to serve as a topic for discussion. Please comment below. We try our best to only post press releases that are associated with peer reviewed scientific literature. Critical discussions of the research are appreciated. If you need help finding a link to the original article, please contact us on twitter or via e-mail.

This press release has been viewed 32 time(s).

Source: http://www.labspaces.net/126027/Transplanted_neural_stem_cells_treat_ALS_in_mouse_model

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